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Institution
Universität Leipzig
Medizinische Fakultät
Department für Operative Medizin
Forschungslaboratorien der Chirurgischen Kliniken I und II
Address
Liebigstraße 20
04103 Leipzig
Deutschland
GERiT
This institution in GERiT
04103 Leipzig
Projects
Research Grants
Completed projects
Cellular and molecular mechanisms of the improvement of non-alcoholic steatohepatitis by mesenchymal stem cells in the immune-deficient mouse
(Applicant
Christ, Bruno
)
Der Proteinase-aktivierte Rezeptor 2 in mesenchymalen Stammzellen - Bedeutung für die Entwicklung und Progression des hepatozelluären Karzinoms
(Applicants
Christ, Bruno
;
Kaufmann, Roland
)
Die Rolle von CD97 in der Entstehung, Invasion und Metastasierung kolorektaler Karzinome (CD97 - elucidating the role in colorectal carcinoma development, invasion and metastasis)
(Applicant
Aust, Gabriela
)
Experimental and clinical proof-of-concept to establish stem cell treatment of post-hepatectomy liver failure
(Applicants
Christ, Bruno
;
Tautenhahn, Hans-Michael
)
Functional characterization of CD97 in intestinal epithelial cells
(Applicants
Amasheh, Salah
;
Aust, Gabriela
)
Role of CD97/ADGRE5 in allergic asthma
(Applicants
Aust, Gabriela
;
Polte, Tobias
)
Role of chemokines and chemokine receptors in the selective migration of leukocytes in autoimmune thyroid diseases
(Applicant
Aust, Gabriela
)
Verbesserung des akuten Leberversagens durch hepatozytär differenzierte mesenchymale Stammzellen im autologen (syngenen) Rattenmodell
(Applicant
Christ, Bruno
)
Research Units
Current projects
FOR 5151: Quantifying Liver Perfusion-Function Relationship in Complex Resection - A Systems Medicine Approach (QuaLiPerF)
(Spokesperson
Dahmen, Uta
)
P1 - Metabolic profiling of the hepatic sinusoid
(Applicant
Christ, Bruno
)
Completed projects
Evaluation of CD97 signaling in transgenic mice
(Applicant
Aust, Gabriela
)
FOR 2149: Elucidation of Adhesion-GPCR Signalling
(Spokesperson
Langenhan, Tobias
)
Collaborative Research Centres
Completed projects
Molekulare Mechanismen der durch die proinflammatorischen Cytikone IL6, IL1ß und TNFa vermittelten Hemmung der glucagon- und insulinabhängigen Genexpression von Stoffwechselenzymen
(Project Head
Christ, Bruno
)
Additional Information
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